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Simultaneously, SIRT1 can deacetylate nuclear factor erythroid 2-related factor 2 (Nrf2), promote its binding to the nuclear antioxidant response element, thereby enhancing the transcription of downstream antioxidant molecules ( 4.3 Anti-apoptotic effects Studies have shown that apoptosis is one of the key pathological features of epilepsy, particularly DRE (83)
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